**1. Introduction**

DAVFs are arteriovenous shunts from a dural arterial supply to a dural venous channel, typically supplied by pachy-meningeal arteries and located near a major venous sinus. The etiology of these lesions is not fully understood; some are congenital, and others are acquired. DAVFs in the pediatric population are associated with structural venous abnormalities [1], but most DAVFs are thought to be acquired. Different etiologies have been implicated in this phenomenon, namely sinus thrombosis, trauma, or surgery [2–6].

Yasargill noted that Rizzoli, in 1881, was the first to describe an arteriovenous malformation (AVM) that involved the dura mater and Sachs reported the first, angiographic description in 1931. Subsequently cranial dural fistulas have been most frequently described at the transverse sinus and cavernous sinus, although they occur at every cranial dural sinus. Dural arteriovenous fistulas (DAVFs) can occur anywhere within the intracranial dura mater. DAVFs are rare vascular abnormalities. They consist of numerous tiny connections between branches of dural arteries and veins or a venous sinus [7]. The true incidence of DAVFs is unknown [8]. However, the reported incidence of intracranial DAVFs is approximately 10–15% of all intracranial vascular abnormalities. A good percentage of DAVFs are asymptomatic or undergo spontaneous involution therefore the true incidence may be much more [9]. DAVFs are acquired lesions; that is why they present later in life than AVMs [10].
