**Chapter 8**

*Inflammatory Heart Diseases*

2001;**1**:181-193

2009;**120**:1266-1286

2007;**3**:853-876

diabetic rats. Acta Physiologica, Pharmacologica et Therapeutica Latinoamericana. 1998;**48**(2):59-63 independent risk factor for ischemic heart disease [see comments]. The New England Journal of Medicine.

[62] Huang JP, Huang LM. Insulin Resistance and Cardiomyopathy, Cardiomyopathies. Taiwan: Chang

[63] Wright JJ, Kim J, Buchanan J, Boudina S, Sena S, Bakirtzi K, et al. Mechanisms for increased myocardial fatty acid utilization following shortterm high-fat feeding. Cardiovascular

Research. 2009;**82**(2):351-360

1996;**334**(15):952-957

Gung University; 2012

[54] Cai L, Kang YJ. Oxidative stress and diabetic cardiomyopathy: A brief review. Cardiovascular Toxicology.

[55] Xu J, Zou MH. Molecular insights and therapeutic targets for diabetic endothelial dysfunction. Circulation.

[56] Hadi HA, Suwaidi JA. Endothelial dysfunction in diabetes mellitus. Vascular Health and Risk Management.

[57] Tang M, Zhang W, Jiang H, Dai H, Zhang Y. High glucose promotes the production of collagen types I and III by cardiac fibroblasts through a pathway dependent on extracellular-

Molecular and Cellular Biochemistry.

[58] Kajstura J, Fiordaliso F, Andreoli AM, Li B, Chimenti S, Medow MS, et al. IGF-1 overexpression inhibits the development of diabetic cardiomyopathy and angiotensin II-mediated oxidative stress. Diabetes.

[59] Michaelson J, Hariharan V, Huang H. Hyperglycemic and hyperlipidemic

biomechanical properties. Biophysical Journal. 2014;**106**(11):2322-2329

BV. Electrocardiographic repolarization complexity and abnormality predict all-cause and cardiovascular mortality in diabetes: The strong heart study. Diabetes. 2004;**53**(2):434-440

[61] Despres JP, Lamarche B, Mauriege P, Cantin B, Dagenais GR, Moorjani S, et al. Hyperinsulinemia as an

signal-regulated kinase 1/2.

2007;**301**(1):109-114

2001;**50**(6):1414-1424

conditions alter cardiac cell

[60] Okin PM, Devereux RB, Lee ET, Galloway JM, Howard

**108**
