**5. Conclusion**

Dyslipidemia induced by stress is part of the body's response to cope with stressors. The mobilization of lipids, glucose and proteins, allows the organs and tissues to maintain homeostasis and adapt to the stressor. Any deficiency in the activation of this mobilization of energetic fuels can compromise the survival of the individual. Therefore, the increase in blood lipids induced by stress is adaptive and it should return to normal levels when the stressor ends. However, when the stressor is maintained over a long period, the dyslipidemia induced by stress persists and may have deleterious effects, contributing to the occurrence of insulin resistance, obesity, hypertension and atherosclerosis. Considering that physical inactivity may potentiate these effects, the association of physical exercise and control of hypercaloric food consumption have been used in the treatment of dyslipidemia. Knowledge about the physiological mechanisms involved in the adaptive role of transient dyslipidemia induced by acute stress, and in the deleterious effects of sustained dyslipidemia induced by chronic stress is very important in the improvement and development of preventive and therapeutic approaches because in modern society we are continuously exposed to stressors.

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