**1. Introduction**

Most pregnant women have vaginal discharges that are either physiologic or pathologic. The challenge to the clinician is to separate the vaginal infections with potentially serious input for pregnancy from annoying but not serious secretions, irritation and pruritus [1]. Infectious vaginitis is usually caused by yeast, such as *Trichomonas vaginalis*, bacterial vaginosis, gonor‐ rhoea, *Chlamydia trachomatis*, *Mycoplasma*, Group B streptococcus or herpes [1]. Normal vaginal secretions consist of water, electrolytes, epithelial cells, microbial organisms, fatty acid and carbohydrate compounds [1, 2].The concentration of anaerobic bacteria is usually five times than that of aerobic organisms. The most prevalent organisms in the vagina are lactobacilli, *Streptococci*, *Staphylococcus epidermidis*, *Gadnerella vaginalis* and *Escherichia coli*. Anaerobic species that are frequently isolated include *Peptostreptococci*, anaerobic *lactobacilli* and *bacter‐ oides* [3].

Vaginal pH, glycogen content and amount of secretion influence the quantity and type of organisms present in the vagina. Lactobacilli restrict the growth of other organisms by producing lactic acid, thus maintaining a low pH. These organisms also produce hydrogen peroxide, which is toxic to anaerobes. The normal vaginal bacterial population assists in inhibiting the growth of pathologic vaginal organisms. If the normal vaginal ecosystem is altered, there is a greater chance of proliferation of pathogenic organisms. The challenge of treating vaginitis in pregnancy is the necessity of making accurate diagnosis and treating correctly [2]. True infections (some of which can have dangerous effect on gestation) must be separated and distinguished from the exaggeration of physiologic discharge by pregnan‐ cy. Infection with bacterial vaginosis, *Chlamydia trichomonas* or Group B Streptococcus has been associated with septic abortion, premature rupture of membranes and premature delivery [2, 4].
