**6. Conclusion**

All of the results mentioned above show that the MDA-MB-231 cells likely display a mesenchymal-like phenotype that facilitates the cells to be a highly metastatic breast cancer cell line. However, a deeper understanding of the cell morphology, gene expression and intracellular mechanisms and pathways of the cancer cells that can explain the interaction between mesenchymal-like and epithelial cells in the MDA-MB-231 cells is warranted. By targeting this phenotype, the metastatic potency and the growth of the cancer cells may be controlled or effectively reduced. A potential anticancer drug can also be identified to treat both human breast cancers and other malignancies. Perhaps, withdrawing the progenitor factor from a tumour may serve as a potential machinery target in cell-mediated therapy for human breast cancer.
