**9. Conclusion**

There are limited studies about cognitive dysfunction in COVID-19 disease in the literature. However, the negative effects of the pandemic have decreased. There is some evidence about the long-term cognitive effects of the COVID-19 disease in the literature. Therefore, it is necessary to plan more investigations about cognitive dysfunction and COVID-19 disease. Understanding the etiopathogenesis is important for diagnosis and treatment modality.

All results demonstrate that it is not possible to explain "cognitive COVID-19" with a single scenario. Etiopathogenesis has multifactorial effects. The disease has a complex nature. Understanding the disease and its treatment is not easy. This complex interaction makes it difficult to determine a single cause or effect. Understanding the etiopathogenesis of this process will decrease the risk of "cognitive COVID-19." It is known that most of this process is at the micromolecular part with inflammatory mechanisms. In addition, interactions at the receptor level are also important in this process. Therefore, determination of the etiopathogenetic process is essential for detection of treatment options.
