**1. Introduction**

Pituitary adenomas (PA) are benign tumors which account for being the second most common intracranial tumors after meningiomas [1]. The incidence of PA is 4.36 per 100,000 and can affect all age groups [1]. However, PA is uncommon in the 1st decade of life with a prevalence of 1–10% when compared to all brain tumors in that age group [2]. The overall chance of developing a pituitary adenoma increases with age, and the non-secretory type is most common after 40 years old [2, 3]. Presentation is highly dependent on the whether the tumor is capable to disrupt hormone homeostasis. Secretory adenomas, also called "functional" adenomas, tend to present early in the clinical course of disease. Conversely, non-secreting adenomas, also called non-functional" adenomas, typically present after reaching a critical size, leading to compression of surrounding neuronal and/or vascular structures.

The first step in the management of a patient with a pituitary adenoma is to distinguish the lesion between a secreting and a non-secreting one. The secretingtype subclassification is based on the specific hormone release by the tumor. Despite advancements in pharmacologic and radiotherapeutic management, surgery is still considered the main modality of treatment for most pituitary adenomas.
