**8. Conclusion**

Ovarian cancer causes substantial morbidity and mortality. Owing to unspecific signs at the early stage of the disease, their appearance at an advanced stage, and poor survival, the difficulty of promptly diagnosing ovarian cancer at its early stage remains difficult. Improved methods of detection are, therefore, urgently needed. This chapter identifies the possible clinical usefulness of epigenetic signatures such as DNA methylation, modifications of histones, and microRNA dysregulation, which play an essential role in ovarian carcinogenesis and its use in the development of diagnosis, prognosis, and biomarkers for prediction. New treatment options separate from conventional treatment options chemotherapy that benefits from developments in the understanding of ovarian cancer pathophysiology to enhance performance, they are required. Recent work has shown that mutations in epigenetic regulator-encoding genes are mutated in ovarian cancer, driving tumorigenesis and drug resistance. Several of these modifiers of epigenetics for ovarian cancer treatment have emerged as promising drug targets.
