**Author details**

Ngoc-Anh Le Biomarker Core Laboratory, Foundation for Atlanta Veterans Education and Research, Atlanta Veterans Affairs Health Care System, Atlanta, Georgia, USA

\*Address all correspondence to: anh.le@va.gov

© 2020 The Author(s). Licensee IntechOpen. This chapter is distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/ by/3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

**113**

*Postprandial Triglycerides, Oxidative Stress, and Inflammation*

expression of toll-like receptor-4 and suppressor of cytokine signaling-3. Diabetes Care. 2010;**33**:991-997

[9] Wang L, Guo L, Zhang L, Zhou Y, He Q, Zhang Z. Effects of glucose load and nateglinide intervention on endothelial function and oxidative stress. Journal Diabetes Research.

[10] Ceriello A, Taboga C, Tonutti L, et al. Evidence for an independent

postprandial hypertriglyceridemia and hyperglycemia on endothelial dysfunction and oxidative stress generation. Effects of short- and longterm simvastatin treatment. Circulation.

2013;**2013**:849295

and cumulative effect of

2002;**106**:1211-1218

2005;**338**:668-676

[11] Niki E, Yoshida Y, Saito Y, Noguchi N. Lipid peroxidation: Mechanisms, inhibition, and biological effects. Biochemical and Biophysical Research Communications.

[12] Liu T, Stern A, Roberts L, Morrow J. The isoprostanes: Novel prostaglandin-like products of the free radical catalyzed peroxidation of arachidonic acid. Journal of Biomedical

[13] Milne G, Musiek E, Morrow J. F2 isoprostanes as markers of oxidative stress in vivo: An overview. Biomarkers.

[14] Esterbauer H, Schaur R, Zollner H. Chemistry and biochemistry of 4-hydroxynonenal, malondialdehyde and related aldehydes. Free Radical Biology and Medicine. 1991;**11**:81-128

[15] Holvoet P, Perez G, Zhao Z. MDAmodified LDL in patients with atherosclerotic disease. The Journal of Clinical Investigation.

Science. 1999;**6**:226-235

2005;**10**(Suppl 1):S10-S23

1995;**95**:2611-2619

*DOI: http://dx.doi.org/10.5772/intechopen.91303*

[2] Camps J, editor. Oxidative Stress and Inflammation in Non-Communicable Diseases—Molecular Mechanisms and Perspectives in Therapeutics. Switzerland: Springer; 2014

[3] Que X, Hung M, Yeang C. Oxidized phospholipids are proinflammatory

hypercholesterolaemic mice. Nature.

[5] Bonnefont-Rousselot D, Bastard JP, Jaudon MC, Delattre J. Consequences of the diabetic status on the oxidant/ antioxidant balance. Diabetes & Metabolism. 2000;**26**:163-176

[6] Piconi L, Quagliaro R, Assaloni R, et al. Constant and intermitent high glucose enhances epithelial apoptosis through mitochondrial superoxide overproduction. Diabetes/Metabolism Research and Reviews. 2006;**22**:198-203

[7] Vries Md KB, Janssen H, Njo T, Westerman E, Cabezas MC. Postprandial inflammation: Targeting glucose and lipids. Advances in Experimental Medicine and Biology.

[8] Deopurkar R, Ghanim H, Friedman J, Abuaysheh S, Sia C, Mahanty P. Differential effects of cream, glucose, and orange juice on inflammation, endotoxin, and the

2014;**824**:161-170

[1] Ebrahim S, Pearce N, Smeeth L, Casas J, Jaffar S, Piot P. Tackling noncommunicable diseases in low- and middle-income countries: Is the evidence from high income countries

all we need? PLoS Medicine.

2013;**10**:e1001377

and proatherogenic in

2018;**558**(7709):301-306

[4] Catherwood M, Powell L, Anderson P, McMaster C, Sharpe P, Trimble E. Glucose-induced oxidative stress in mesangial cells. Kidney International. 2002;**61**:599-608

**References**

*Postprandial Triglycerides, Oxidative Stress, and Inflammation DOI: http://dx.doi.org/10.5772/intechopen.91303*
